Cholesterol no longer a risk factor for heart disease. Look to CRP?

Dr. James Stein, MD from the University of Wisconsin Medical School in Madison, praised the JUPITER study for exposing the fact that current therapeutic LDL-cholesterol levels are not only arbitrary, but are in fact a poor indicator of cardiovascular disease (CVD) risk. ?Most patients with heart attacks have normal cholesterol values,? he stated. With the cholesterol theory crumbling the industry is under intense pressure to come up with a new risk factor, and one that can be treated with the same statin drugs they have invested so much money in. Enter Dr. Ridker and C-reactive protein (CRP). Ridker has been pushing treating CRP with statins for years. But is CRP a risk factor? A National Panel on CRP found no evidence treating CRP levels will improve survival rates (www.urmc.rochester.edu/pr/News/story.cfm?id=182). Elevated CRP levels are associated with many things including; anger, stress, arthritis, cancer, lupus, pneumonia, TB, oral contraceptive use, pregnancy, heart attacks, surgery, trauma, intense exercise, etc. It?s a marker for disease, not the cause. But since statins lower CRP levels slightly, you can count on CRP becoming the new cholesterol. The public will be made to fear CRP, be tested for it, and be put on dangerous statins to lower it. What a racket.

  

Saturated fat is no villian.

Saturated fat found mainly in animal products has been vilified by physicians, the media, and the edible oil industry for over 60 years, despite mounds of evidence to the contrary. A meta-analysis of 21 prospective epidemiologic studies that had a total of 347,747 participants, showed that there is no significant evidence for concluding that dietary saturated fat is associated with an increased risk of cardiovascular disease or stroke.

Saturated fats have been nourishing societies around the world for thousands of years. If animal fats (saturated fats) are so dangerous, and vegetable oils (polyunsaturated fat) are so healthy, why are we so unhealthy as a nation? The scientific data of the past and present does not support the assertion that saturated fats cause heart disease. As a matter of fact, people who have had a heart attack haven?t eaten any more saturated fat than other people, and the degree of atherosclerosis at autopsy is unrelated to diet.Ravnskov, Uffe. ?The cholesterol Myths: Myth number 4?

  

Lower fat means higher CVD risk

Coronary heart disease is associated with diet. Nutritional recommendations are frequently provided, but few long term studies on the effect of food choices on heart disease are available. We followed coronary heart disease morbidity and mortality in a cohort of 1752 rural men participating in a prospective observational study. Dietary choices were assessed at baseline with a food questionnaire. 138 men were hospitalized or deceased owing to coronary heart disease during the 12 year follow-up. Daily intake of fruit and vegetables was associated with a lower risk of coronary heart disease when combined with a high dairy fat consumption, but not when combined with a low dairy fat consumption. Consuming wholemeal bread or eating fish at least twice a week showed no association with the outcome.

Food Choices and Coronary Heart Disease: A Population Based Cohort Study of Rural Swedish Men with 12 Years of Follow-up

  

Eat less salt and increase your risk of heart disease.

We are continuously told to reduce our sodium intake by eating a low sodium diet. Dr.s, dietitians, and nutritionists insist it’s good for our cardiovascular system especially if one has cardiovascular disease. But is it?

Salt induced hypertension, despite what you’ve heard, is very uncommon. More-over, the vast majority of people who switch to a diet low in sodium have no change in blood pressure and may be doing more harm than good. According to a cohort study of 7154 individuals, sodium is inversely related to cardiovascular mortality.

After adjusting for variables, low sodium consumption was associated with a 37% greater risk for cardiovascular disease mortality, and 28% increased risk of all-cause mortality.

  

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